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doi: 10.1016/j.leukres.2013.07.021 151 LangemeijerSde SwartLYuGSmithACrouchSJohnstonTet al
The two largest clusters contained 18 and 4 animals (Fig
Microglial NF-B activation under GSH-depleted conditions creates a pro-inflammatory loop where increased ROS promotes NF-B activation, which upregulates inflammatory cytokines and further stimulates oxidative stress (Liu et al., 2017
Extrapolating from tendon data and mechanisms suggests bone healing likely benefits though direct confirmation limited
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