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When this process is inhibited it can result in accumulated proteins, toxins, and inflammatory metabolic byproducts in the brain that may contribute to dementia

Skene, N

In the TCEPreduced samples, mmPEG 24 was added to a final concentration of 10 mM

Reactive astrocytes: the nexus of pathological and clinical hallmarks of Alzheimers disease

Since this could lead to lipid peroxidation, we measured the production of 4-hydroxynonenal (4HNE), which results from the oxidation of membrane lipids

The first one includes soluble dimeric enzymes localized mainly in cytosole but certain members of this superfamily have been also identified in mitochondria (Robinson et al., 2004) or in peroxisomes (Morel et al., 2004)