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Indirect Evidence for A Role of Redox Dysregulation in Als As well as the studies described above, there is also evidence that cellular processes associated with redox homeostasis are dysregulated in ALS (Figure 2)

The conjugation of glutathione on the previously hydrophobic-toxic substrate results in a soluble compound, which is more readily exocytosed by the cell

In vivo data were analysed with a two-way ANOVA followed by Tukeys post-hoc test (fasted glucose levels, ipGTT glucose excursion, body weight and food intake) or a one-way ANOVA followed by Dunnetts or Tukeys post-hoc test (glucose AUC, terminal fat and clinical chemistry data)

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While they often have normal or even elevated bone mineral density (BMD), they paradoxically experience higher fracture rates