In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis
Vitamin B12 is a vitamin essential for many bodily functions, including energy production, brain function, and the formation of red blood cells
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2 Read our extended information on the Treatment of Pernicious Anaemia: Treatment of Pernicious Anaemia NICE Guideline: Vitamin B12 deficiency in over 16s: diagnosis and management March 2024 Devalia V, Hamilton M, Molloy A