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Given that major surgery can induce T-cell immunometabolic paralysis linked to mitochondrial damage, mitochondria-supportive interventions may also be mechanistically relevant to postoperative immunosuppression ( 3.1.3.2 Anesthetic agents as immunometabolic modulators The drugs we use routinely in anesthesia can be leveraged as immunometabolic modulators

DSS-induced MPMs impaired MCECs migration, while GHK-Cu augmented this capacity (Figures 4E,F)

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When miR-34a was overexpressed in HNGC-2 cells, cell proliferation and cell cycle progression were decreased, while caspase-dependent apoptosis was increased, as compared to control cells

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