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Int J Med Microbiol 302:187194

This may be due to the existence of an intact IGF1R in both WT and knockin mice as well as effects from both ligands, insulin and IGF-1, which could at least partially compensate for their downstream signaling effects

Enzyme Inhib

[11] Polymerase chain reaction-restriction fragment length polymorphism analyses were used for the genotyping of MTHFR polymorphisms

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N-methylated glutathione analogues demonstrate a 16.1-fold increase in oral bioavailability compared to native GSH