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Engaging all three pathways simultaneously drives glucose-dependent insulin secretion through the two incretin arms while adding glucagon-receptor agonism implicated in energy expenditure, the pharmacological rationale that distinguishes it from single (GLP-1 (Sema)) and dual (GLP-2 (Triz)) incretin agonists

The DDIT4 gene with known inhibitory role of mTOR was remethylated after BCG, a step likely to allow improved glucose uptake

However, AKI-mediated effects in the liver have not been comprehensively assessed

Best for small doses during early escalation

R., Heilmann, C., Hoogwerf, B

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