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In ALS models, SOD1 mutations do not markedly alter astrocytic baseline states but lower the threshold for reactivity, which sensitizes astrocytes to oxidative, glutamatergic and inflammatory stress and drives exaggerated reactive responses to otherwise moderate insults 80

doi: 10.1016/j.nut.2013.01.001 27 FanJLiGWuLTaoSWangWShengZet al

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The receptor antagonist findings Gastroprotective effects in stress-induced ulceration models were blocked by haloperidol (alpha-1A and dopamine receptor antagonist), phentolamine (non-selective alpha-adrenergic antagonist) and clonidine (alpha-2A adrenergic agent), but were not affected by prazosin, domperidone or yohimbine

Epithelial-mesenchymal transitions in development and disease