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Conformational changes in tyrosine 11 of neurotensin are required to activate the neurotensin receptor 1

First, we investigated the presence of GLP-1R expression in several pancreatic cancer cell lines as well as in normal pancreatic cells by qRT-PCR and western blot analysis (Fig

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249 The key molecular event of KD treatment in promoting immunotherapy is the activation of AMPK, which phosphorylates PD-L1, resulting in its disrupted interaction with CMTM4 and subsequent PD-L1 degradation, which phosphorylates EZH2, resulting in improper PRC2 function and subsequently enhanced expression of IFN and antigen-presentation gene