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Definitive evidence of the role of FFAs in GH-mediated insulin resistance was obtained in clinical studies in which the effects of exogenously administered GH on insulin resistance were abrogated by Acipimoxan inhibitor of lipolysis [185, 190, 191]
DSIP flux across the BMEC monolayers was also found to be bidirectional, insensitive to metabolic inhibitors, and not altered by high concentrations of tryptophan
It also covers an important point most pages skip: the two compounds have been studied separately far more than they have been studied together
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