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In animal models of atherosclerosis, the GLP1 analogue exendin 4 substantially reduced the accumulation of monocytes and macrophages in the vascular wall and inhibited atherogenesis by regulating inflammation in macrophages 95

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"I have seen patients move through the transition

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Peak levels of the peptide are achieved in tissue approximately 1 hour after administration and then slowly decline over the next 48 hours [1]