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As NAD+ declines with age, sirtuin activity is reduced, compromising all downstream functions
Benoist CC, Wright JW, Zhu M, Appleyard SM, Wayman GA, et al
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It activates the HGF/c-Met receptor pathway in the brain, promoting the formation of new synaptic connections (synaptogenesis)
The catalog includes a wide range of regulatory peptides, signaling fragments, and synthetic analogs used in vascular biology, metabolic signaling, and cellular pathway research
The wound healing cascade and GHK-Cu: Normal wound healing occurs in four overlapping phases: Hemostasis clotting Inflammation immune cell recruitment Proliferation new tissue formation Remodeling scar maturation GHK-Cu appears to favorably influence phases 2, 3, and 4 simultaneously: In the inflammatory phase , it reduces pro-inflammatory cytokines (IL-6, TNF-) while preserving necessary immune signaling In the proliferative phase , it stimulates keratinocyte migration and fibroblast proliferation, accelerating epithelial coverage In the remodeling phase , it promotes type I collagen deposition over type III (scar) collagen, leading to better scar outcomes This multi-phase tissue repair activity makes GHK-Cu a compound of interest when studied alongside other regenerative peptides
