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This may have been caused by senescence-related cellular deteriorations, leading to the substantial metabolic or physiological changes that significantly affect the overall metabolic energy consumption

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However, during sepsis, ETC dysfunction leads to electron leakage and disproportionate ROS formation, resulting in the collapse of mitochondrial membrane potential, impaired ATP synthesis, and activation of mitochondrial-mediated apoptosis (Nagar et al., 2018)