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Since the discovery of ferroptosis, much solid evidence has been obtained in experimental tumor models that ferroptosis has good anti-cancer effects

There are theoretical concerns, like a potential connection to cancer, that can't be dismissed without more research

By using these advanced treatments, we can give patients personalized and effective care

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Given that some mutant enzymes retain low-level FAD-binding capacity, a gene + high-dose riboflavin synergy strategy has been proposed: in AIFM1 encephalopathies, low-level AAV transduction re-establishes baseline protein expression, while daily riboflavin doses above 400 mg act as a molecular chaperone to stabilize FAD binding a regimen shown to improve ICARS scores and neuroimaging outcomes (54, 75)

Skipping site rotation Alternate injection sites to avoid tissue damage