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Malignant cells exhibit marked elevation of redox regulators including Trx1 and GSH, creating therapeutic barriers by neutralizing ROS-mediated cytotoxicity and impairing Fenton reaction-driven ferroptosis (34,69)

Conversion of senescent cartilage into a pro-chondrogenic microenvironment with antibody-functionalized copper sulfate nanoparticles for efficient osteoarthritis therapy

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The mechanism of action of FOXO4-DRI lies in its ability to competitively interfere with the binding of endogenous FOXO4 to the p53 proteina central regulator of the cell cycle and apoptosis