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Additionally, aging-induced mutations in mtDNA further aggravate mitochondrial dysfunction by disrupting the synthesis of critical proteins involved in the OXPHOS system, crucial for energy metabolism [116]
D.et al (2015)
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It plays a pivotal role in energy balance and postprandial metabolism by enhancing glucose-dependent insulin secretion, suppressing glucagon release, delaying gastric emptying, and promoting satiety through central mechanisms [8, 9]
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