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Nitric oxide scavenging by red blood cell microparticles and cell-free hemoglobin as a mechanism for the red cell storage lesion

Its expression is essential for proper axonal growth and repair, and mutations in GAP43 result in axonal atrophy

Specifically, glutamate and cysteine complex to make gamma-glutamylcysteine,

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Inadequate utilization of vitamin B 12

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