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Preclinical results in murine models treated with GLP-1RAs suggest that the potential effects of semaglutide in AD may be exerted via impact on several AD-related pathophysiologic processes including neuroinflammatory pathways, vascular and BBB integrity, reduced synaptic loss, and neuroprotection [19, 21, 23,24,25,26, 46,47,48]
Remind yourself that this side effect is well-documented, temporary in most cases, and not indicative of serious harm
Additionally, semaglutide slows gastric emptying and reduces appetite, which can lead to unintended undereating and subsequent dehydration or electrolyte imbalance
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