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Shi Y, Li H, Chu D, Lin W, Wang X, Wu Y, et al
A829968 132
In Alzheimers disease, NLRP3 inflammasome has emerged as a pathological component participating in the impaired removal of amyloid- by microglia (Lonnemann et al., 2020)
It is distinguished in the research literature by its selectivity: published pre-clinical work reports it stimulates GH release without significantly elevating cortisol, prolactin, or ACTH, unlike earlier secretagogues in its class
Association between serum concentrations of persistent organic pollutants and prevalence of newly diagnosed hypertension: results from the National Health and Nutrition Examination Survey 1999-2002
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