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After the knockout of the GLP-1R gene, in transgenic mice expressing human GLP-1R in pancreatic islets and pancreatic ductal cells restored cAMP and serine threonine kinase (Akt) phosphorylation levels in isolated islets, increased GLP-1-stimulated insulin secretion and GLP-1R-dependent -cell proliferation, and promoted transgenic mice for glucose regulation in response to feeding (Lamont et al., 2012)

specializes in pharmaceutical development with emphasis on technical expertise and therapeutic optimization

Presented for research literature review only Ganther HE (1971)

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