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Because typical early atherosclerotic lesion formation observed in hyperhomocysteinemic animals can be prevented by an antiplatelet therapy with dipyridamole, the authors concluded that platelet-mediated proliferation of smooth muscle cells is a potential cellular mechanism involved in the atherogenicity of hyperhomocysteinemia
It's crucial for the development and function of key immune cells like neutrophils and T-cells
It also regulates your immune response and protects against oxidative stress like free radicals
Presented for research literature review only Feng D et al
Fragrance is so soothing, almost spa-like
It also affected mitochondrial function, including changes in membrane potential
