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That gap is real, and it is worth resolving before you try to reconcile two sources

Other mechanisms of acetylcarnitine action Acetylcarnitine has been suggested to protect against oxidative stress through the induction of antioxidant genes and an increase in HO-1 (heme oxygenase 1) expression in vitro and in vivo [17, 18]

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The decline in oestrogen appears to remove a layer of protection, which in turn accelerates depletion of this critical molecule, creating a cycle that worsens symptoms

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