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doi: 10.1016/j.ejps.2012.03.010 209 HassanAASayyahSG

We synthesized multiple peptides that varied in staple length and position

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In accordance with its reduced capacity for stimulating insulin release, exendin-asp3 failed to reduce apoptosis following glucolipotoxicity 24 or endoplasmic reticulum stress 25 , both involved in T2D pathogenesis

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