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They can support mechanistic hypotheses about endothelial response and microvascular repair, but they do not establish human wound healing efficacy, cardiovascular benefit, clinical safety, or suitability for any condition where abnormal angiogenesis could be biologically relevant
This process functions to limit growth and protein synthesis when nutrient availability is compromised
Research suggests it may work through different mechanisms, potentially involving direct interaction with adipocyte membranes or intracellular signaling pathways distinct from classical GH receptor activation
therefore, nutritional deficiency of this vitamin is rare in adults in the absence of malabsorption disorders
Critical roles in angiogenesis (VEGF) and cellular signaling pathways
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