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These pathways not only preserve proteome integrity but also offer powerful therapeutic opportunities, as demonstrated by the emergence of proteasome inhibitors, autophagy modulators, and molecular chaperone-targeting agents in clinical medicine (Tables 1 and 2)
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Searching for ferroptosis inhibitors or inducers has become a hot research topic in recent years, but due to the intricate regulatory mechanisms of ferroptosis and individual differences of patients, the targeted therapy of ferroptosis still stays in the preliminary stage
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Through Nrf2 signaling and immune modulation, the ozone-oxygen therapy stimulates glutathione production and nudges the mitochondrial function back to normal
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