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Inflammation in the CNS increases the risk of AD initiation and exacerbates the amyloid and tau pathology and the cognitive decline [12]

The findings strongly suggested that the mitochondria had some sort of way to detect and adjust these fluctuating levels

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Lipoic acid decreases lipid peroxidation and protein glycosylation and increases (Na(+) + K(+))- and Ca(++)-ATPase activities in high glucose- treated human erythrocytes