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Classical exhaustion is typically driven by prolonged antigenic stimulation and is characterized by progressive loss of effector function, sustained expression of multiple inhibitory receptors (including programmed cell death protein 1, PD-1), andcriticallya stable exhaustion-associated epigenetic landscape that constrains reinvigoration even after checkpoint blockade ( A key unresolved question is whether perioperative immune dysfunction engages the core transcriptional circuitry of exhaustion

It may be relevant for studies examining cellular aging, structural protein turnover, and oxidative signaling pathways under controlled conditions

Material that tests below specification, lacks identity confirmation, or carries undocumented impurities undermines reproducibility