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Once the microglia engulf the senescent neurons, they become senescent, dysfunctional and amplify pathogenesis through inflammation, releasing toxic tau and failure to survey the brain microenvironment

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Gastrointestinal cytoprotection: characterized in NSAID-, ethanol-, stress- and colitis-induced injury models, with rapid restoration of mucosal integrity and barrier function [1]

Pro-inflammatory mediators such as TNF-, IL-1, and IL-17A enhance SMCs responsiveness to growth factors by upregulating PDGFR expression, thereby promoting SMC proliferation and contributing to intestinal wall thickening and fibrosis 85

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Some may do so, but it is usually not recommended by reputable clinicians