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Beyond their reactivity in stroke, astrocytes also exhibit a form of autophagy plasticity, dynamically adjusting autophagic activity in response to pathological stimuli such as A accumulation
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By simultaneously pushing the accelerator via GHRH receptors and removing the brakes via somatostatin inhibition, the resulting pulse is significantly more potent than either peptide could produce in isolation
These findings support further evaluation in the ongoing phase 3 programme
They involve different immune pathways, different target tissues, and different disease mechanisms
Cognition, 12.9 and 15.0
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