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Recently, SLC1A3, as a crucial effector of p53, has been shown to support cell survival and growth in the absence of glutamine 149

In effect, adding prandial RAIs to the treatment regimen mimics the physiological meal-simulated insulin release, which is lost first as beta-cell function declines

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J Neurochem 109(Suppl 1):189197 Hassel B, Sonnewald U, Fonnum F (1995) Glial-neuronal interactions as studied by cerebral metabolism of [2-13C]acetate and [1-13C]glucose: an ex vivo 13 C NMR spectroscopic study

Preclinical data suggest GIP receptor engagement modulates lipid partitioning pathways and may act synergistically with GLP-1 receptor signalling in energy-balance models