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Qiu WM, et al

While our study did not employ organelle-specific ROS probes to pinpoint the precise origin of ROS, the combined evidence- significant total ROS accumulation and the finding that ER stress activation is a prerequisite for mitochondrial apoptosis- strongly suggests the existence of an ER-to-mitochondria signaling axis in NBDHEX-induced cell death

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Normalizing tumor vasculature to reduce hypoxia, enhance perfusion, and optimize therapy uptake

The injection site in the documentation determines the code, not the drug