Therefore, enhanced glutaminolysis might eventually contribute to the initiation and progression of cancer by stimulating cell growth via the mTORC1 pathway and enhancing aerobic glycolysis while disrupting the proper elimination of misfolded proteins, damaged DNA and organelles through the inhibition of autophagy and the DNA damage response 166
doi:10.1002/14651858.CD002081
Copper (cu) induced changes of lipid metabolism through oxidative stress-mediated autophagy and Nrf2/PPAR pathways
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