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For example, Parkinsons and Alzheimers diseases have high levels of oxidative stress and damage to the brain with correspondingly low active glutathione (GSH) levels
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The rationale is dual-pathway activation: CJC-1295 (GHRH analog) activates the GHRH receptor to stimulate GH synthesis and baseline release Ipamorelin (ghrelin mimetic) activates GHS-R1a to amplify GH pulse amplitude By engaging both regulatory pathways simultaneously, researchers have observed synergistic GH elevation greater than either compound alone
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