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Almost 20 years ago, a high extracellular level of glutamate was described as inhibitor for cystine uptake and inductor of a specific cell death termed oxytosis (44)

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TTP treatment lowered SBP to a similar extent as the clinical standard captopril, although the duration of the antihypertensive effect was shorter in the TTP group

Specifically, glutamate inhibits paravascular movement though the NMDA receptor and facilitates interstitial fluid clearance though the AMPA/kainate receptor while GABA does not influence paravascular movement but accelerates interstitial fluid drainage