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However, in the posterior cortex, CAT activity was reduced in the VPA+LipRSV group compared to the VPA group, indicating that LipRSV treatment may have negatively modulated the enzymes expression or activity, either by attenuating oxidative stress and thus reducing enzymatic defense needs, or through a possible pro-oxidant effect dependent on the region and experimental conditions

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Notably, PUFAs in neuronal membranes are primary targets for lipid peroxidation, with ROS derived from mitochondrial metabolism and NADPH oxidases further amplifying oxidative stress (28) (Figure 3)

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