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A primary challenge lies in SFNs suboptimal pharmacokinetic profile, characterized by rapid metabolism, limited oral bioavailability, and chemical instability
The resulting Gly-His-Lys-Cu(II) complex shifts the peptide's behavior from a passive ligand to an active modulator of intracellular copper trafficking [2]
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If anxiety occurs, it is more likely during the initial weeks as the body adjusts to the medication
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Thus, we attempted to solve the complex structure with a more stable product compound, AsA, using active site mutant protein (C20S)
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