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(2023) observed no difference between GIP- and GLP-1-stimulated cAMP production or insulin release from INS-1 832/3 cells despite reduced G s recruitment to GIPR
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Thus, by promoting intimal hyperplasia, these vascular disorders (which are related to smooth muscle cell proliferation, collagen accumulation, and the extracellular matrix modification that may result from homocysteinylation of the vascular wall structure leading to connective tissue deterioration) might contribute to the atherogenic tendency of hyperhomocysteinemia
MalekiVJafari-VayghanHKashaniAMoradiFVajdiMKheirouriSet al
33 (37), e2102054
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