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It crosses the blood-brain barrier via a saturable transport mechanism, supporting peripheral administration efficacy

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Although the predominance of available literature from in vivo ischemic studies has focused on the involvement of synaptic vesicle zinc, it is likely that intracellular zinc accumulation during ischemia is achieved through a possible synergism of zinc release from both synaptic and intracellular stores of zinc, as most recently demonstrated during global ischemia (38) and reported in other experimental paradigms (4551)

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Rats received a regular diet with 49.39% of energy derived from carbohydrates, 31.67% from protein, and 18.94% from fat