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Additionally, in vitro experiments revealed that ICA inhibits the increase in CTGF and p-Drp1 Ser616 in a dose-dependent manner, while increasing p-Drp1 Ser637, MFN1, MFN2, TFAM, and ATP6

However, each member of this family operates via a distinct mechanism and exhibits specific sites of action in maintaining redox homeostasis [46]

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Preclinical (mostly rat) studies show BPC-157 acts like a supercharged project manager that: Builds new blood vessels super fast (angiogenesis) It switches on the VEGF system (the same one your body uses to grow new capillaries) and boosts nitric oxide so fresh blood, oxygen, and nutrients flood the injured area

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PI3K/Akt, Nrf2/ARE, and NF-B, which regulate inflammation, oxidative stress, and tissue regeneration (Liu et al