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We treated cells with both iHAP1 [16], a PP2A agonist, and LB-100 [17], an inhibitor of PP2A, and showed that iHAP1 mediated the activation of PP2A mitigated hypoxia-induced neuronal death (Supplemental Fig
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Daily observations were made during bandage changes, noting the presence of exudate and monitoring the progression of healing until Day 21
Mol Cell Biochem
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