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3 Mechanisms of sulforaphane-based neuroprotection 3.1 Sulforaphane and oxidative stress regulation Reactive oxygen species (ROS), such as superoxide anions and hydrogen peroxide, are byproducts of cellular metabolism and function within organelles such as mitochondria, endoplasmic reticulum, and peroxisomes (Oswald et al., 2018
There were cured jaundice, weight loss, liver enlargement, microscopy (i.e., piecemeal necrosis, focal lytic necrosis, apoptosis and focal inflammation, and disturbed cell proliferation (Ki-67-staining), cytoskeletal structure in the hepatic stellate cell (-SMA staining), collagen presentation (Mallory staining), and biochemistry presentation (Sever et al., 2019), and counteracted the increased NOS3 expression, interleukin (IL)-6, tumor necrosis factor (TNF)-, and IL-1 levels
Transcriptome analysis of hepsin / and wild-type mice during the early period after APAP exposure To clarify the function of hepsin in the early stages of APAP-induced hepatotoxicity, a transcriptome analysis employing RNA sequencing was conducted within the first 2 h post-APAP administration
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