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Within the context of concurrent GLP-1R and GIPR agonism, however, preclinical energy-expenditure models suggest that GCGR engagement may contribute to resting metabolic rate modulation and fatty-acid oxidation in hepatic cell lines
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Carcinogenesis 12 , 14711475 (1991)
Activation of mTORC1 by leucine is potentiated by branched-chain amino acids and even more so by essential amino acids following resistance exercise
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