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Presented for research literature review only LaCount DW et al

It is well established that cap-independent translation of ATF4 requires eIF2, which is activated by endoplasmic reticulum (ER), oxidative, or mitochondrial stress 42,43,44

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These treatment conditions were chosen to mimic excess heme from chronic hemolysis and systemic hypoxic conditions that occur in SCD patients 1,2,11

Researchers should ensure all handling and use complies with relevant laboratory guidelines and standards

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