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I'm old enough to remember when leptin was going to be the key to all of this

doi: 10.1111/j.2040-1124.2010.00022.x 125 KleinertMSachsSHabeggerKMHofmannSMMllerTDKleinertMet al

It is proposed that interface architecture, tether composition, organelle positioning and mitochondrial vesicle flux collectively determine whether activation driven stress is limited and resolved or propagated into persistent immune dysfunction

However, landmark analysis, adjusting the cohort selection period to 2, 6, 9, and 12 months post-transplant, demonstrated consistent beneficial effects of GLP-1 RAs on all-cause mortality, MACEs, and MAKEs (Supplemental Table 13)

Hirsch, et al., 2018

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