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Sakurabayashi T, Takaesu Y, Haginoshita S et al (1999) Improvement of myocardial fatty acid metabolism through L-carnitine administration to chronic hemodialysis patients
The two are frequently blended because their mechanisms are independent and additive in published GH-axis models
Conversely, CLEC4E-induced robust nitric oxide release was mediated by enhanced translation of key enzymes via p38-dependent EIF5A hypusination, thereby inhibiting NLRP3-dependent IL-1 production (Fig
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This approach is especially valuable as we continue to refine diagnostic criteria for clinical obesity
Prevention of HGF nitration-dysfunction offers novel potential to suppress the progression of age-related disorders of muscle homeostasis including muscle atrophy and impaired regeneration accompanied by up-regulation of intramuscular fibrogenesis (fibrosis) and adipogenesis (fat infiltration)
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