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and HIF1a flox/flox mice, The Jackson Laboratory, RRID: IMSR_JAX:007,561) mice were used for birth dated experiments
Gap junction dysfunction reduces acetaminophen hepatotoxicity with impact on apoptotic signaling and connexin 43 protein induction in rat
We observed that at early stages of tumour progression, NKX2-1 functions as a tumor suppressor, but later becomes an aggressive oncogene , contributing to cancer progression when nutrients such as serine and glycine become scarce
P., Stiner, J., Makarov, M
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