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The synthetic triterpenoid CDDO-Me blocks survival of neoplastic mast cells and synergizes with KIT D816V-targeting drugs in inducing growth inhibition

These effects seem to be mediated by GLUT4, which triggers PI3K/PKB, and glucose, which inhibits the LKB1/AMPK pathway

It could be that researchers just havent figured out the right doses, protocols or combinations of GLP-1-based molecules

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That evidence is not currently available for a broad declaration that liposomal or S-acetyl glutathione is clinically superior