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Effect of SEMA on SIRT1/AMPK signaling pathway in mice with TAA-induced liver fibrosis The hepatic SIRT1 levels as well as the p-AMPK protein expression were remarkably reduced in the TAA-treated group compared to the control group values, as shown in Fig

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Cancer cell biology research examining IGF1R-driven proliferation often uses LR3 as a standard reagent precisely because the dose-response across a multi-day proliferation assay is more interpretable when the ligand is stable in the media

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The values are expressed as the mean standard error of the mean