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To identify glutathione-overproducing mutants, acrolein was used as a selective agent, given glutathiones key role in protecting cells from acrolein-induced toxicity (Mano et al., 2009
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Presented for research literature review only Li HT et al
This actin-sequestering action drives coordinated angiogenesis and shifts macrophage polarization from pro-inflammatory M1 toward anti-inflammatory M2 phenotypes, both processes that become less efficient as estrogen levels decline during perimenopause
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