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Helicobacter (2021) 26(3):e12796
Foundational research also supports the role of microglia in A uptake and degradation, mediated by specific receptors and enzymatic pathways, alongside associated neuroinflammatory responses observed in AD pathology (Lee and Landreth, 2010)
glp 1 GLP-1
Our identification of VRK2-mediated SLC7A11 trafficking suggests a potential immune evasion mechanism, as SLC7A11-high tumors often display T cell exclusion phenotypes [37], warranting future investigation into whether VRK2 expression could predict therapeutic response to combined ferroptosis inducers and immune checkpoint inhibitors
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